Respiratory syncytial virus (RSV) is the major respiratory pathogen of infants and children worldwide, with no
effective treatment or vaccine available. Steady progress has been made in understanding the respiratory syncytial virus
lifecycle and the consequences of infection, but some areas of RSV still remain poorly understood. Although many of the
interactions between virus proteins that are required for efficient RSV assembly have been elucidated, many questions still
remain regarding viral assembly, as well as the mechanisms of RSV budding. This review will summarise the current understanding
of RSV assembly, including the various interactions between virus proteins and the involvement of cellular
factors with a view to identifying possible attenuation and/or drug targets within the assembly pathway.