Login Register Cart 0
Generic placeholder image

Current Pharmaceutical Design

Editor-in-Chief

ISSN (Print): 1381-6128
ISSN (Online): 1873-4286

Back
Journal Home About Journal Editorial Board Journal Insight Current Issue Volumes/Issues
Subscribe
Editorial

Oxidative Medicine in Brain Injury

Author(s): Matilde Otero-Losada and Francisco Capani

Volume 25, Issue 45, 2019

Page: [4735 - 4736] Pages: 2

DOI: 10.2174/138161282545200110113639

Next »
[1]
Potts MB, Koh SE, Whetstone WD, et al. Traumatic injury to the immature brain: inflammation, oxidative injury, and iron-mediated damage as potential therapeutic targets. NeuroRx 2006; 3: 143-53.
[2]
Youdim MBH. Monoamine oxidase inhibitors, and iron chelators in depressive illness and neurodegenerative diseases. J Neural Transm (Vienna) 2018; 125: 1719-33.
[3]
Barnham KJ, McKinstry WJ, Multhaup G, et al. Structure of the Alzheimer’s disease amyloid precursor protein copper binding domain. A regulator of neuronal copper homeostasis. J Biol Chem 2003; 278: 17401-7.
[4]
Morton PD, Dellarole A, Theus MH, Walters WM, Berge SS, Bethea JR. Activation of NF-κB in Schwann cells is dispensable for myelination in vivo. J Neurosci 2013; 33(24): 9932-6.
[5]
Brambilla R, Bracchi-Ricard V, Hu WH, et al. Inhibition of astroglial nuclear factor kappa B reduces inflammation and improves functional recovery after spinal cord injury. J Exp Med 2005; 202(1): 145-56.
[6]
Kimura N, Tokunaga C, Dalal S, et al. A possible linkage between AMP-activated protein kinase (AMPK) and mammalian target of rapamycin (mTOR) signalling pathway. Genes Cells 2003; 8: 65-79.
[7]
Malik AR, Urbanska M, Skalecka A, Jaworski J. Beyond control of protein translation: what we have learned about the non-canonical regulation and function of mammalian target of rapamycin (mTOR). Biochim Biophys Acta 2013; 1638: 1434-48.
[8]
Kanekiyo T, Bu G. The low-density lipoprotein receptor-related protein 1 and amyloid-beta clearance in Alzheimer’s disease. Front Aging Neurosci 2014; 6: 93.
[9]
Reddy PH. Amyloid precursor protein-mediated free radicals and oxidative damage: implications for the development and progression of Alzheimer’s disease. J Neurochem 2006; 96: 1-13.
[10]
Díaz A, Treviño S, Guevara J, et al. Energy drink administration in combination with alcohol causes an inflammatory response and oxidative stress in the hippocampus and temporal cortex of rats. Oxid Med Cell Longev 2016; 2016: 8725354.
[11]
Oyenihi OR, Afolabi BA, Oyenihi AB, Ogunmokun OJ, Oguntibeju OO. Hepato- and neuro-protective effects of watermelon juice on acute ethanol-induced oxidative stress in rats. Toxicol Rep 2016; 3: 288-94.
[12]
Stroev SA, Tjulkova EI, Gluschenko TS, Rybnikova EA, Samoilov MO, Pelto-Huikko M. The augmentation of brain thioredoxin-1 expression after severe hypobaric hypoxia by the preconditioning in rats. Neurosci Lett 2004; 370: 224-9.
[13]
Gan Y, Ji X, Hu X, et al. Transgenic overexpression of peroxiredoxin-2 attenuates ischemic neuronal injury via suppression of a redox-sensitive pro-death signaling pathway. Antioxid Redox Signal 2012; 17: 719-32.
[14]
Hu X, Weng Z, Chu CT, et al. Peroxiredoxin-2 protects against 6-hydroxydopamine-induced dopaminergic neurodegeneration via attenuation of the apoptosis signal-regulating kinase (ASK1) signaling cascade. J Neurosci 2011; 31: 247-61.
[15]
Caruso C, Durand D, Schioth HB, et al. Activation of melanocortin 4 receptors reduces the inflammatory response and prevents apoptosis induced by lipopolysaccharide and interferon gamma in astrocytes. Endocrinology 2007; 148: 4918-26.
[16]
Delgado R, Carlin A, Airaghi L, et al. Melanocortin peptides inhibit production of proinflammatory cytokines and nitric oxide by activated microglia. J Leukoc Biol 1998; 63: 740-5.

Rights & Permissions Print Cite
Article Metrics
5
World Health Day
© 2024 Bentham Science Publishers | Privacy Policy